SuicidDentistV1, Main, Exploration, bibRecord, 000642

Health hazards and nitrous oxide: a time for reappraisal.

Identifieur interne : 000642 ( Main/Exploration ); précédent : 000641; suivant : 000643

Health hazards and nitrous oxide: a time for reappraisal.

Auteurs : J A Yagiela [États-Unis]

Source :

Anesthesia progress [ 0003-3006 ]

RBID : pubmed:1809046

Descripteurs français

KwdFr :
- 5-Methyltetrahydrofolate-homocysteine s-methyltransferase (antagonistes et inhibiteurs), Anesthésie dentaire (MeSH), Animaux (MeSH), Dentistes (MeSH), Exposition professionnelle (MeSH), Humains (MeSH), Polluants atmosphériques d'origine professionnelle (MeSH), Protoxyde d'azote (effets indésirables), Troubles liés à une substance (MeSH), Vitamine B12 (métabolisme), États-Unis (MeSH).
MESH :
- antagonistes et inhibiteurs : 5-Methyltetrahydrofolate-homocysteine s-methyltransferase.
- effets indésirables : Protoxyde d'azote.
- métabolisme : Vitamine B12.
- Anesthésie dentaire, Animaux, Dentistes, Exposition professionnelle, Humains, Polluants atmosphériques d'origine professionnelle, Troubles liés à une substance, États-Unis.
Wicri :
- geographic : États-Unis.

English descriptors

KwdEn :
- 5-Methyltetrahydrofolate-Homocysteine S-Methyltransferase (antagonists & inhibitors), Air Pollutants, Occupational (MeSH), Anesthesia, Dental (MeSH), Animals (MeSH), Dentists (MeSH), Humans (MeSH), National Institute for Occupational Safety and Health, U.S. (MeSH), Nitrous Oxide (adverse effects), Occupational Exposure (MeSH), Substance-Related Disorders (MeSH), United States (MeSH), Vitamin B 12 (metabolism).
MESH :
- chemical , adverse effects : Nitrous Oxide.
- chemical , antagonists & inhibitors : 5-Methyltetrahydrofolate-Homocysteine S-Methyltransferase.
- chemical , metabolism : Vitamin B 12.
- chemical : Air Pollutants, Occupational.
- geographic : United States.
- Anesthesia, Dental, Animals, Dentists, Humans, National Institute for Occupational Safety and Health, U.S., Occupational Exposure, Substance-Related Disorders.

Abstract

Recent adoption by the American Conference of Governmental Industrial Hygienists of a Threshold Limit Value of 50 ppm for an 8-hour average exposure to nitrous oxide (N2O) increases the likelihood for its regulation by state and federal occupational health agencies. This review outlines current information on the health risks of N2O inhalation to provide a basis from which safe and reasonably attainable exposure limits can be proposed. Although N2O was for many years believed to have no toxicity other than that associated with its anesthetic action, bone marrow depression in patients administered N2O for extended periods of time and neurological abnormalities in health care workers who inhaled N2O recreationally have disproved this notion. Retrospective surveys of dental and medical personnel have also linked occupational exposure to N2O with a number of health problems and reproductive derangements. Nitrous oxide reacts with the reduced form of vitamin B12, thereby inhibiting the action of methionine synthase, an enzyme that indirectly supports methylation reactions and nucleic acid synthesis. Many, if not all, of the nonanesthetic-related adverse effects of N2O may be ascribed to this action. Animal and human studies indicate that the toxic effects of N2O are concentration- and time-dependent. It is suggested that a time-weighted average of 100 ppm for an 8-hour workday and/or a time-weighted average of 400 ppm per anesthetic administration would provide adequate protection of dental personnel and be achievable with existing pollution control methods.

PubMed: 1809046
PubMed Central: PMC2162364

Affiliations:

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Le document en format XML

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<front><div type="abstract" xml:lang="en">Recent adoption by the American Conference of Governmental Industrial Hygienists of a Threshold Limit Value of 50 ppm for an 8-hour average exposure to nitrous oxide (N2O) increases the likelihood for its regulation by state and federal occupational health agencies. This review outlines current information on the health risks of N2O inhalation to provide a basis from which safe and reasonably attainable exposure limits can be proposed. Although N2O was for many years believed to have no toxicity other than that associated with its anesthetic action, bone marrow depression in patients administered N2O for extended periods of time and neurological abnormalities in health care workers who inhaled N2O recreationally have disproved this notion. Retrospective surveys of dental and medical personnel have also linked occupational exposure to N2O with a number of health problems and reproductive derangements. Nitrous oxide reacts with the reduced form of vitamin B12, thereby inhibiting the action of methionine synthase, an enzyme that indirectly supports methylation reactions and nucleic acid synthesis. Many, if not all, of the nonanesthetic-related adverse effects of N2O may be ascribed to this action. Animal and human studies indicate that the toxic effects of N2O are concentration- and time-dependent. It is suggested that a time-weighted average of 100 ppm for an 8-hour workday and/or a time-weighted average of 400 ppm per anesthetic administration would provide adequate protection of dental personnel and be achievable with existing pollution control methods.</div>
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	Serveur d'exploration sur le suicide chez les dentistes
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Serveur d'exploration sur le suicide chez les dentistes

Health hazards and nitrous oxide: a time for reappraisal.

Health hazards and nitrous oxide: a time for reappraisal.

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